Koul (2000) undertook a study to look for patterns of non-convulsive status epilepticus (NCSE) in 12 affected children. Changes in the EEG of continuous or asynchromous 1.5-2 Hz spike and slow wave discharges with background slowing was taken as confirmation for the presence of Lennox Gastaut epilepsy. Three patients (two boys aged 2 and 4-years, and a girl, aged 7 years) were diagnosed with the condition. The two older children had shown tonic clonic and myoclonic seizures before the onset of NCSE. The youngest boy, however, had shown tonic clonic and myoclonic astatic seizures. CT and/or MRI scans in two of the patients were normal, while in the older male, it revealed cerebellar atrophy. All three patients were initially administered an antiepileptic and later put on a benzodiazepine derivative when it failed to control the fragmentary seizures. The outcome remained poor in all three cases.

In 2006, Koul et al. analyzed data from the pediatric neurology department at Sultan Qaboos University Hospital for children undergoing evaluation for developmental delay and epilepsy. Corpus callosum agenesis was the most common neuronal migrational disorder, with 22 patients (55%). There were 14 boys and 8 girls. Eight children (four boys and four girls) in this group had epilepsy. These were infantile spasms in four, generalized tonic-clonic seizures in two, and Lennox-Gastaut syndrome in two.

Yaqub (1993) analyzed electroclinical seizures observed by long video split-screen recording in 21 patients with Lennox-Gastaut syndrome (LGS). All patients had atypical absence seizures, 18 (81%) had tonic seizures, and 4 (21%) had myoclonic-atonic seizures. Tonic seizures were axial with flexion or extension of the head or trunk, or global with generalized tonic spasm mimicking infantile spasm, or involved the eyeballs only (either brief, with upward deviation of eyeballs or long, with oscillatory nystagmus). EEG showed either a bilateral 10-13-Hz rhythm or generalized synchronous spike wave at 3 Hz. Myoclonic-atonic seizures involving limbs, trunk, or neck were either brief or massive; the discharges were 2-3.5-Hz spike wave. Atypical absence seizures evolved gradually, terminated abruptly, and manifested alone or with subtle motor activity or oral automatism. EEG discharges were variable and of different types: (a) Diffuse irregular spike wave at 2-2.5 Hz with or without fragmentation (consciousness was regained during fragmentation or when spike wave discharges were < 2 Hz), (b) irregular diffuse fast activity at 10-13 Hz, or (c) a combination of fast spike wave or sharp waves of increasing amplitude followed by synchronous spike wave discharges at 3 Hz.