Foveal Hypoplasia 1

Alternative Names

  • FVH1
  • Foveal Hypoplasia 1 with or without Anterior Segment Anomalies and/or Cataract
  • Foveal Hypoplasia-Presenile Cataract Syndrome
  • O'Donnell-Pappas Syndrome

Associated Genes

Paired Box Gene 6
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WHO-ICD-10 version:2010

Diseases of the eye and adnexa

Disorders of sclera, cornea, iris and ciliary body

OMIM Number

136520

Mode of Inheritance

Autosomal dominant

Gene Map Locus

11p13

Description

Foveal hypoplasia has generally been associated with other disorders such as aniridia, ocular albinism, microphthalmia, retinopathy of prematurity, incontinentia pigmenti, myopia or achromatopsia. However, recent studies have uncovered rare cases of isolated foveal hypoplasia. Isolated foveal hypoplasia is a disorder characterized by the lack of foveal depression and the absence of foveal pigmentation or the foveal avascular zone. Patients with FVH1 usually have low visual acuity and congenital nystagmus.

FVH1 is diagnosed by optical coherence tomography (OCT). There are currently no treatment options available besides cataract surgery for patients with significant lens opacity.

Several cases of isolated foveal hypoplasia have been associated with mutations in the Pax6 gene and have been shown to follow an autosomal dominant pattern of inheritance. Pax6 is a transcriptional factor involved in ocular morphogenesis. Genetic defects in Pax6 have generally been linked with aniridia, albeit certain mutations were only associated with FVH1. These include a heterozygous 799C-T mutation in exon 7 resulting in R125C missense mutation; a heterozygous 553G-T transition, predicted to result in G64V substitution; and a heterozygous gene splice mutation (IVS4 + 5G>C) resulting in a mutant mRNA lacking exon 4. However, a recent study has reported a case of isolated foveal hypoplasia with no detected gene defects in Pax6 and a pattern of inheritance that is seemingly autosomal recessive.

Molecular Genetics

Several cases of isolated foveal hypoplasia have been associated with mutations in the Pax6 gene and have been shown to follow an autosomal dominant pattern of inheritance. Pax6 is a transcriptional factor involved in ocular morphogenesis. Genetic defects in Pax6 have generally been linked with aniridia, albeit certain mutations were only associated with FVH1. These include a heterozygous 799C-T mutation in exon 7 resulting in R125C missense mutation; a heterozygous 553G-T transition, predicted to result in G64V substitution; and a heterozygous gene splice mutation (IVS4 + 5G>C) resulting in a mutant mRNA lacking exon 4.

However, a recent study has reported a case of isolated foveal hypoplasia with no detected gene defects in Pax6 and a pattern of inheritance that is seemingly autosomal recessive.

Epidemiology in the Arab World

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Other Reports

Saudi Arabia

[See also: Sudan > Al-Saleh et al. (2011)]

Sudan

Al-Saleh et al. (2011) reported the case of a 7 year old Sudanese boy presenting with poor vision since childhood. The patient’s parents were first cousins with no family history of similar symptoms. Eye investigation found normal anterior segments and ruled out ocular albinism and aniridia. The patient showed nystagmus and the fovea was found to be absent in both eyes. Persistent inner retinal layers within the fovea and an absence of foveal depression were revealed by optical coherence tomography. The patient, his parents and unaffected sibling were screened for mutations in the GPR143, PAX6 and OCA2 genes. Mutations in GPR143 and OCA2 have been associated with ocular albinism while mutations in PAX6 result in aniridia. No mutations were discovered in any of the three genes. A subsequent screen also failed to find any chromosomal abnormalities. The patient was diagnosed as having isolated foveal hypoplasia with the genetic cause being undetermined. Unlike previously reported studies on FVH1, this study postulates an autosomal recessive pattern of inheritance owing to the parents being consanguineous and unaffected.

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